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These findings suggest that selenomethyl selenocysteine may enhance the responsiveness of tumor tissues to anticancer agents by reducing HIF-1α expression in mice bearing tumors, thereby potentially improving the efficacy of cancer treatments (151)
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The cholesterol-rich remaining particles (now referred to as chylomicron remnants), return apo C-II molecule to HDL and are recognized by specific hepatic apo E and apo B-100 receptors that rapidly remove them from the circulation by endocytosis
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